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Consistently, we found that the cardiac glycoside drugs ouabain, digitoxin and digoxin, which competitively inhibit binding of spike protein to ACE2 29 , were able to rescue spike-induced loss of cAMP-activated CFTR channel activity and CFTR protein expression

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In addition, fbf-1 fbf-2 mutants raised at 25 possessed distal germ cells in mitotic metaphase or anaphase, consistent with active divisions (Figure S1A)