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Enhanced insulin secretion in CD38-expressing transgenic mice

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(2018) using inducible pluripotent stem cells (iPSC)-derived neurons with triplication of SNCA gene showed that oligomeric -synuclein through inhibition of mitochondrial complex I activity caused oxidation of a subunit of ATP synthase leading to the opening of mitochondrial permeability transition pore with consequent mitochondrial swelling and cell death