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2010;91(5):S147883

ADMINISTRATION: Intramuscular, intravenous or subcutaneous

Eck HP, Gander H, Hartmann M, et al

Jamroz, D

Secondly, hyperglycemia-driven mitochondrial dysfunction promotes the accumulation of the TCA cycle intermediate succinate, which inhibits prolyl hydroxylases (PHDs) and stabilizes HIF-1, a master transcriptional regulator that enhances the expression of IL-1 and other M1-associated inflammatory mediators, creating a feed-forward inflammatory loop (Tannahill et al., 2013

Preclinical studies have validated the safety of Nrf2 activators, laying the groundwork for their clinical application in OA