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It is possible that the recovery of glutathione levels may enhance the survival of affected neurons, either by increasing synthesis of GSH or by slowing its degradation [25]

Reconstituted solutions should be protected from reducing agents (such as DTT or beta-mercaptoethanol), which can cleave the disulfide bond and disrupt the active conformation

Effects of chronic ethanol feeding on rat hepatocytic glutathione

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Further, overproduction of free radicals have also been shown to occur due to spontaneous breakdown (autoxidation) and/or excessive metabolism by monoamine oxidase of adrenaline, noradrenaline and dopamine, which can further overwhelm the brains antioxidant systems (Cohen, 1984

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