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Molecular mechanisms in the pathogenesis of diabetic nephropathy: an update

doi:10.1002/14651858.CD012357.pub2 42 Chen, C.-C

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Isogenic mutants deficient in the activities of thioredoxin (54), catalase (KatA) (55), NADPH quinone reductase (56), and superoxide dismutase (46) are sensitive to host colonization and susceptible to oxidative damage

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