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In summation, GGCs potential to elevate intracellular glutathione levels above homeostasis in an energy-efficient manner, makes it a promising candidate as a potential therapeutic for mitochondrial diseases

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Figure 3 illustrates how the network between the gene/pathways and compound targets was built using Cytoscape 3.10.0

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Jan 3, 2026 Understanding what happens when stopping GHK-Cu requires examining the peptide's mechanism of action influencing collagen synthesis, skin regeneration, antioxidant effects, and anti-inflammatory properties to determine whether these changes represent temporary stimulation requiring ongoing supplementation or permanent improvements persisting after treatment cessation

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