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As this does not mirror the clinical efficacy observed for efgartigimod within CIDP, and as the used FcRn antagonist did reduce total levels of circulating IgG to an extent beyond that observed for IVIg treatment, it must be because of this animal model being driven by other mechanisms than solely autoreactive IgG

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The IOM and NHS/BNF recommend the above amounts as the overall dietary supply

By disrupting FOXO4-p53 interactions, it may encourage the clearance of damaged cells and restore tissue function

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