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TB-500 contributes to tendon repair through a different and complementary pathway

References 1 Journal of Pharmacology and Experimental Therapeutics (PubMed)McCoy et al

Because it avoids those receptors, AOD 9604 does not elevate IGF-1 levels, does not affect blood sugar regulation, and does not carry the risks associated with exogenous growth hormone use

10.1073/pnas.191189098 369 LiJ.UverskyV

This likely reflects the balancing effects of GLP-1 and GIP receptor activation, including glucose-dependent insulin secretion and reduced energy intake

Modulation of these molecular mechanisms leads to a significant inhibition of cell viability, colony formation, and cell migration, ultimately contributing to apoptosis and increased sensitivity to radiation therapy 30 (Figure 3)