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BPC-157 has been examined in similar models for its apparent role in modulating cell migration and adhesion, suggesting that combined investigation may provide insight into coordinated cytoskeletal and extracellular interactions

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Cuproptosis increases the stability of NFE2L2 by suppressing its proteasomal degradation, leading to transcriptional upregulation of GCLC and GCLM and enhanced GSH synthesis, thereby suppressing cuproptosis in pancreatic cancer cells 169

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