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Besides invasion, miR-181-5p suppressed branching-morphogenesis by directly regulating c-Met (Korhan et al., in vitro and in vivo studies have demonstrated that miR-101-3p inhibits HCC progression by targeting HGF and it could be regarded as another potential therapeutic tool for regulating cell proliferation and metastasis (Liu et al., In our review, the potential roles of the HGF/c-Met axis in other hallmarks of HCC, such as interactions with tumor microenvironment and drug resistance, are also highlighted
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