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The selenium appeared to provide a protective effect against oxidative stress and Ca (2+) entry through modulation of neutrophil TRPV1 calcium channel

10.1016/j.jnutbio.2009.01.011 118 WonJ

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The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels