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Evaluating the effects of amantadin, modafinil and acetyl-l-carnitine on fatigue in multiple sclerosis Result of a pilot randomized, blind study

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doi: 10.15406/hij.2018.02.00047 68

The aim of this study is to exploit GSH-responsive AP site capture reagent (AP probe-net), which responses to the elevated glutathione (GSH) levels in the tumor micro-environment (TME), releasing reactive alkoxyamine to trap AP sites and block the APE1-mediated BER for targeted anti-tumor activity against ATC