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Long-chain fatty acids cross the myocardial cell membrane via transporters (such as H-FABP), bind with CoA, and are then converted into acylcarnitine by CPT-1 on the outer mitochondrial membrane to enter the mitochondria for -oxidation ( 3 -induced cardiomyopathy model ( There have been no studies on the mechanism of LC in treating myocarditis

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The specific gene affected determines the specific EDS type