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There is no general agreement about specific pharmacological treatment regimens for uncomplicated NMS

In that case, a combo plan can make sense, but it needs a safety check first

Glucagon-like peptide-1 decreases endogenous amyloid-beta peptide (Abeta) levels and protects hippocampal neurons from death induced by abeta and iron

The glutamine metabolism antagonist, JHU083, has been shown to inhibit M.tb replication both in vitro and in vivo, improving survival, reducing lung bacterial burden, and improving lung histopathology

Its status today is strictly research-only

it could be mild with occasional relapses or severe with long-term issues that cause distress and harm