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On the contrary, like in apoptosis, the cell membrane remains intact in eryptotic cells, so that heme, hemoglobin, and other potential erythrocyte-derived DAMPs are preserved from passive cell membrane destruction-associated liberation [78, 91]

Certain tumors escape ferroptosis by upregulating system xc (a cystine/glutamate antiporter composed of SLC7A11 and SLC3A2) or glutathione peroxidase 4 (GPX4), a key enzyme that reduces lipid hydroperoxides and protects cells from ferroptotic death [1]

Hence, high levels of free radicals can cause damage to them and induce dysraphic anomalies (neural tube defects)