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Notably, the dual role of EMFs manifests in their ability to induce pro-metastatic phenotypes under certain parameters [7,8] while exhibiting therapeutic potential under others [9-12] [Table 1]

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It can simultaneously reduce lung permeability, neutrophil infiltration, pro-inflammatory cytokine synthesis, and pulmonary bacterial load, thereby improving the survival rate of mice (283)

Association between a single nucleotide polymorphism in the 3-UTR of ARHGEF18 and the risk of nonidiopathic pulmonary arterial hypertension in Chinese population