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Although both pharmacological approaches target GLP-1, important differences exist concerning the mode of administration (subcutaneous injection versus oral ingestion), the efficacy (better with GLP-1 agonists), the effects on body weight and systolic blood pressure (diminution with agonists versus neutrality with gliptins), the tolerance profile (nausea and possibly vomiting with agonists) and the cost (higher with GLP-1 receptor agonists)
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58 Glucose-induced pancreatic insulin secretion can be stimulated by treatment with a truncated form of glucagon-like peptide-1, which activates hepatic vagus nerve afferents and, subsequently, vagus nerve efferents innervating the pancreas
Pattern 1: Continue at the therapeutic dose This is what the trial data supports most strongly
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