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Icariin enhances cell survival in lipopolysaccharide-induced synoviocytes by suppressing ferroptosis via the xc-/GPX4 axis

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Protein glycation is thus responsible for the development of inflammation, but it also disrupts extracellular matrix remodeling and intercellular signaling 77,78

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1 for schematic overview of sample collection and processing procedures.) The roles of these preparation conditions and their respective hypothesis-testing comparisons are detailed in Table 1