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Activation of the p53/p21 pathway can enable cancer cells to resist oxidative stress, and this effect may involve inhibition of nucleotide synthesis (Maddocks et al, 2013)

The agency accepted the clarification

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This suggests that chronic lymphocytic leukemia cells were highly active in de novo FA synthesis as well as in FA catabolism 53

Some clues to their distinctions have come from studying chimeric receptors, in which domains within each hemireceptor were exchanged between the IR and IGF-IR 4954

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