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In hepatocarcinogenesis, aflatoxin B1 (AFB1) exposure primarily induces oxidative stress through CYP3A4-mediated metabolic activation, generating ROS and MDA that directly cause DNA damage [389, 390]

ASAIO J

A clearer delineation of these interacting pathways will be important for advancing our understanding of ME/CFS pathophysiology and for informing more mechanism-based therapeutic strategies targeting chronic stress-related cognitive impairment

larger, longitudinal studies are needed to validate the observed ALC-cholesterol association and investigate causality regarding ASCVD risk

doi: 10.1186/gm367

Unveiling the Power of Glutathione: What is Glutathione