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In summary, the antioxidant properties of selenoproteins showed therapeutic potential by counteracting the accumulation of damage induced by oxidative stress in cartilage

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Reduction with TCEP, followed by the addition of excess iodoacetamide (IAM), releases a stoichiometric amount of CAM-S-CAM-TPP from RSS-CAM-TPP and RS(S) n S-CAM-TPP that incorporates the terminal sulfur atom from RSS and RS(S) n S

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This effect is mediated, at least in part, by the suppression of the Toll-like receptor 4 (TLR4)/nuclear factor-kappa B (NF-B) signaling pathway (Zhai et al., 2025)