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Treatment of a Case of Severe Insulin Resistance as a Result of a PIK3R1 Mutation With a Sodium-Glucose Cotransporter 2 Inhibitor

Its ability to cross the bloodbrain barrier (BBB) makes it a crucial therapeutic agent for restoring dopaminergic neurotransmission, thereby influencing motor function, cognition, and neuroprotection

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63 Additionally, ample GSH improves cell viability, whereas its depletion drastically increases susceptibility to ROS-inducing antibiotics

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