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Morris ER, Caswell SJ, Kunzelmann S, Arnold LH, Purkiss AG, Kelly G, et al
Free-radical mechanisms in tissue injury
Semaglutide activated the P38 MAPK signaling pathway To further explore the mechanism by which Semaglutide inhibits the progression of OSCC, we investigated the phosphorylation status of three MAPK signaling pathways (ERK1/2, SAPK/JNK and P38) in Cal27 and HSC4 cells after Semaglutide treatment (0, 5, or 10 mol/L, 48 h) by western blot analysis
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Neovascularization after ischemic injury in the brain improves microperfusion of tissue in the peri-infarct area (157)
Uniprot P36969-2) is constituted by a catalytic tetrad that consists of the following residues: selenocysteine (Sec 46 ), glutamine (Gln 81 ), tryptophan (Trp 136 ) and asparagine (Asn 137 ) [35]
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