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The beneficial antioxidant mechanism is attributable to catechin polyphenols binding to sperm membrane phospholipid head groups, thereby creating a physical barrier against lipid-radical propagation 50,52

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Paradoxically, liver-specific FASN knockout promoted hepatic steatosis in mice on a zero-fat diet [60], in which steatosis developed alongside defective PPAR signaling and with a phenotype that could be corrected by dietary fat or a PPAR agonist [60]

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