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As mentioned, counteracted were neurons destruction in the substantia nigra (MPTP) [69], dopamine vesicle depletion (reserpine) [69], dopamine receptors blockade (neuroleptics) [68,70], dopamine over-release and re-uptake inhibition (amphetamine

Monitoring focuses on glucose trends, kidney/liver context, lipids, and symptoms tied to reduced intake or GI effects

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It is also linked to lower glucagon release after meals, which can improve glycemic stability

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