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Since senescent cells may release a senescence-associated secretory phenotype, the authors suggest that lowering the senescent cell burden may reduce pro-fibrotic signaling in the surrounding cells

This is longer than the typical 4-6 week cycle used for musculoskeletal repair, reflecting the slower pace of GI tissue remodeling

This apparent increase in GH synthesis and release per pulse was accompanied by no change in GH pulse frequency or in levels of IGF-I, IGFBP-3, or GHBR

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