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Insulin-like growth factor I and impaired glucose tolerance

The results of a transcriptomic analysis obtained from a mouse model also revealed that the stimulation of GLP-1 receptor by high doses of exendin-4 upregulates IGF-1R typically in pancreatic islets, thus reinforcing the concept that activating the IGF-1R/IRS-2/Akt pathway mediates the prevention of -cell loss under the control of incretins (40)

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Copper is essential for numerous enzymatic functions in the body, including collagen synthesis, antioxidant defense, and tissue remodeling

So thanks so much for breaking that down for us