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In the lipid-rich environment of the CNS, ferroptosis could therefore play a significant role in secondary damage, the neuron-astrocyte interaction is also critical for maintaining brain lipid metabolism, indicating that neurons may be more sensitive to drastic changes in lipid peroxidation, moreover, lipid peroxidation contributes to secondary damage after SCI and highlights the possible use of lipid metabolism regulator to treat SCI [46,47,48]
GHK-Cu also modulates gene expression through mechanisms that remain incompletely characterized but involve broad transcriptomic changes affecting over 4,000 human genes as identified through Connectivity Map (CMAP) analysis
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The opioid crisis alone justified a generation of skepticism
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