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2012;7(4):e35240

Nisin, an apoptotic bacteriocin, induces ChaC1 activation, calcium influx and cell cycle arrest in the G2 phase, leading to increased apoptosis and decreased cell proliferation in vitro and in vivo (68)

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The vast number of preclinical studies on NAD + modulation via precursors or modulators of NADase enzymes has established a tight functional relationship between NAD + metabolism and mitochondrial homeostasis