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In addition to genetic mutations, dysregulation of signaling pathways is a significant characteristic of GBM pathogenesis

Abstract The rising global burden of metabolic diseases, including obesity, type 2 diabetes, hypertension, and non-alcoholic fatty liver disease, has been closely linked to alterations in gut microbial composition and function, which contribute to systemic metabolic dysregulation

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This mechanism is why slow-release carbohydrates and prebiotic fibers are increasingly positioned to raise endogenous GLP-1 and PYY as a non-drug route to appetite regulation

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