Further investigations of channel characteristics, particularly the dose and voltage dependency of TMX effects, are required to confirm whether these hypotheses are correct
(36) demonstrated that key signaling pathwaysincluding the Nrf2-Keap1 pathway, p53, and Hippo pathwaycontribute to the initiation and progression of atherosclerosis by modulating the expression and activity of ferroptosis-related proteins
doi: 10.1109/JSEN.2014.2343152
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