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The common link is that GLP-1s activate receptors that then work on driving down metabolic risk factors through the following mechanisms: Enhancing glucose dependent insulin secretion Slowing gastric emptying and decreasing appetite Reducing postprandial glucagon and food intake UpToDate 2 : (1) ingestion of food results in (2) release of gastrointestinal peptides (GLP-1 and GIP: glucose-dependent insulinotropic polypeptide, gastric inhibitory peptide) and (3) pancreatic beta cell hormones (insulin and amylin)
Hydration and adverse-effect management Severe nausea, vomiting, diarrhea, constipation, or appetite suppression should not simply be accepted as proof that the medication is working. Tracking strength and function Notice what the body can do, not just what it weighs
Am J Physiol Endocrinol Metab 289(5):E829E838
Hims does offer anti-nausea meds to eligible patients, so either he did not get them or they did not do the job
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