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In osteoblasts specifically, AKT promotes GLUT4 translocation via PI3K signaling to restore glucose uptake under insulin-resistant conditions, securing the carbon substrates needed for collagen glycosylation and ATP-dependent mineralization, a metabolic reprogramming function that extends well beyond the hormone's classical glycemic role

Lipid peroxidation/ antioxidant activity in patients with alopecia areata J Eur Acad Dermatol Venereol

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Increased GSSG or ROS-induced generation of protein thiol radicals both trigger protein glutathionylation

Monitoring for signs of infection is crucial

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