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This measurable deficit provides objective, biochemical proof of the mitochondrial dysfunction that patients experience, validating their symptoms and highlighting the carnitine shuttle as a primary target for therapeutic intervention

For a medication where precise dosing determines outcomes, that gap matters enormously

cruzi has evolved to localize FeSODB2 and nsGPx-I antioxidant enzymes in the glycosomal compartment (Wilkinson et al., 2000, 2002b

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