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Although EDS is fundamentally a genetic collagen disorder with no cure yet, these treatments shift the focus from palliative care to reparative care
Lovastatin and simvastatininhibitors of HMG CoA reductase and cholesterol biosynthesis
[DOI] [PubMed] [Google Scholar] 88.Venezia AC, Guth LM, Sapp RM, et al
Collectively, the UPR pathways are effective for halting protein biosynthesis, enriching protein degradation, producing chaperones to recover protein folding and triggering either autophagy or apoptosis
10.18632/oncotarget.23843 33 SchmidtE
The evaluation of the problem led to a solution, and there have been no recurrences
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