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and CPT1C is expressed in the brain ( CPT1A and CPT1C rather than CPT1B , and we speculate that this may have been because these two isoforms showed lower abundance in cardiomyocytes and were thereby more sensitive

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PTD-DBM is more narrowly targeted, focused specifically on removing CXXC5 suppression of Wnt signaling

Moreover, IgA1-CD89 complexes promoted the binding of sCD89 with TFR1 to induce the expression of transglutaminase 2 (TGase2) on the surface of HMCs, thereby facilitating the upregulation of TFR1 [272]

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