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Compounds like Semax upregulate BDNF to promote synaptic plasticity and neuronal survival

These comparative investigations continue supporting the evolution of peptide-based research models

Besides, our research group has previously reported that the MD2-TLR4 complex not only participates in acute inflammatory responses but also recognizes various metabolism-associated molecular patterns (MAMPs) such as palmitic acid, advanced glycation end products, and oxLDL, thereby mediating chronic inflammation in metabolic diseases 46,47,48,49 Thus, we may guess that BDNF attenuates some metabolic diseases through antagonizing TLR4 and then reducing TLR4-driven chronic inflammation

The development of specific peptide therapies for memory support represents a significant advancement in neurocognitive medicine