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In the study by Altinsoy and Dikmen, researchers determined that an increase in gastrointestinal symptoms was positively correlated with both brain fog symptoms and the severity of brain fog

Based on these findings, it was recognized for the first time that impairment of antioxidant defenses (vitamin E-deficiency) and enhanced levels of PUFAs (substrates for LPO) did not just amplify the injury mechanism but switched cell death signaling typically centered around mitochondrial dysfunction (in mice on normal diets) to one dominated by acute LPO [70]

These results strengthen the evidence we are building and represent meaningful progress for a field in urgent need of new treatment options

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