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As expectedly, both the E155A cFSP1 and E156A hFSP1 mutations obviously reduced the CoQ oxidoreductase activity, especially the E156A hFSP1 mutation is completely inactive in the NADH oxidation assay using CoQ 1 as substrate and cell death assay (Fig

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Others see slower initial progress that accelerates over time

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Hyperuricemia-induced acute kidney injury in the context of chronic kidney disease: A case report