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demonstrated that the GLP-1 analogue lixisenatide decreases atherosclerosis in insulin-resistant Apoe/Irs2+/ mice by promoting macrophage polarization towards an anti-inflammatory M2 phenotype, which was associated with enhanced plaque stability (i.e., thicker fibrous caps, fewer necrotic cores, and decreased inflammatory infiltrates)

First, digestion redirects blood flow from the oral mucosa to the GI tract, reducing the efficiency of sublingual uptake

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Suspected side effects should be reported to the MHRA Yellow Card scheme in addition to informing the clinical trial team