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Adaptive behavior in autism: minimal clinically important differences on the Vineland-II

Hines DJ, Kaplan DL

Network pharmacology and molecular docking suggested CA9 as a putative binding target of ATT-I which was confirmed by CETSA demonstrating increased thermal stability of CA9 upon ATT-I treatment

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Rapamycin addresses cellular senescence in the follicle microenvironment by inhibiting mTOR, while GHK-Cu works on Wnt signaling, inflammation, and androgen receptor sensitivity

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