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Curtis, K

Our findings raise the notion that reductions in key antioxidant mechanisms may be the biological risk factor for the development of konzo, particularly those mediated through pathways involving the glutathione peroxidase family

Furthermore, we observed that ACTL6A suppression sensitized GC cells to H 2 O 2 , erastin, and buthionine sulfoximine (BSO), which are ferroptosis inducers, but not to compounds (e.g., etoposide and doxorubicin) that inhibit cell growth through other mechanisms (Fig

Effects of dapagliflozin on development and progression of kidney disease in patients with type 2 diabetes: an analysis from the DECLARE-TIMI 58 randomised trial

Moreover, LPS-induced stimulation of macrophages activates the JAK-STAT1 pathway through the release of IFN-, resulting in the expression of iNOS