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Depending on the disease state of the population under study (e.g., preclinical, symptomatic, late-stage disease) and the objective of the intervention (prevention versus treatment), efficacy can be measured in different ways and commonly relies on the use of measures related to cognitive and functional outcomes, as well as changes in markers of pathobiology

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For example, in atherosclerosis, lipid peroxidation and iron deposition in plaques contribute to lesion instability [379], whereas in acute myocardial infarction ferroptosis plays a direct role in cardiomyocyte cell death during reperfusion

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