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Sustained caloric restriction, even when clinically intended, can lead to micronutrient insufficiencies that compound fatigue, cognitive fog, and muscle loss side effects that are often misattributed to the medication itself

The bioinformatics platform we built for caffeine and EGCG in NASH disease found that the two drugs may greatly overlap in improving the mechanism related to NASH inflammation

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Intracellular Mycobacterium tuberculosis exploits host-derived fatty acids to limit metabolic stress

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These cytokines upregulate TfR1 and downregulate FTH1, thereby promoting intracellular iron overload and ROS generation, which directly sensitize NPCs to ferroptotic death (50)