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5.2 Channelopathies and synaptopathies: molecular roots of Vmes dysfunction MTNs sustain rhythmic bursting through Nav1.6-dependent I NaP , I NaP , and I h currents

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However, their possible contributions toward cardiac function impairment in HF patients are not completely known

The short versions are found in hemolymph, cuticle and fatbody cells, while the long forms are mostly expressed in hemocytes (154)

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