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Role of the alpha-cell in GSIS Proglucagon-derived products signal through the beta-cell GLP-1 receptor and glucagon receptor to potentiate GSIS Evaluation of the mechanisms by which beta-cell GLP-1R activation potentiates insulin secretion offers insight into the propensity for alpha-cell-derived proglucagon products to modulate beta-cell function

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There is no published stability data for the two held together in solution over time