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set out to determine whether enteroendocrine cells (EECs) could sense circulating LPS and thus release GLP-1 in order to modulate the pro-inflammatory response6

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GLP-1 agonists benefit MASH patients by reducing liver fat, decreasing inflammation, and hence improving liver tissue structure, potentially slowing the progression of the disease

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Moreover, the DPP-4-generated metabolites of GLP-1 (Figure 1), lacking the His-Ala dipeptide, have 100-fold less affinity towards GLP-1R

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