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It inhibits nicotinamide N-methyltransferase (NNMT), an enzyme that when overactive, consumes excessive NAD+ and impairs cellular energy metabolism

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The increased presence of the lipopolysaccharide (LPS) component of the bacterial outer membrane results in pro-inflammatory cytokines being released across the bloodbrain barrier to induce central nervous system (CNS) inflammation

This association underscores a possible shared etiological landscape beyond mere comorbidity

It demonstrates promising methods for overcoming mucous and alveolar macrophage absorption deep in the lungs