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The copper chelation that gives GHK-Cu its regenerative mechanism (stimulating collagen type I synthesis, enhancing fibroblast proliferation, and reducing MMP-1 expression) depends on maintaining the exact peptide-copper complex structure

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doi: 10.1158/1535-7163.MCT-09-0161

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We also found that PI3KAkt and MAPK signaling pathways were enriched, consistent with the results of GO analysis