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Key molecular features include the F23 Cagri residue anchoring the peptide at the receptor transmembrane (TM) bundle level and the micelle, an E14-R17 intramolecular salt bridge enhancing helical stability, and C-terminal P37 Cagri interaction with the receptor ECD

All procedural interventions received formal authorization from the Nanjing University of Chinese Medicine Animal Use Committee (No

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MEK blockade converts AML differentiating response to retinoic acid (RA) into extensive apoptosis